维甲酸诱导Ⅰ型基因,retinoic acid-inducible gene-Ⅰ
1)retinoic acid-inducible gene-Ⅰ维甲酸诱导Ⅰ型基因
1.Objective To study the regulatory effect of interferons(IFNs) onretinoic acid-inducible gene-Ⅰ(RIG-Ⅰ) and the roles of RIG-Ⅰ in IFNs signaling pathway.目的研究干扰素(IFNs)对维甲酸诱导Ⅰ型基因(RIG-Ⅰ)的调控作用,同时探讨RIG-Ⅰ对IFNs生物学效应的介导作用。
2)retinoic acid-induced gene Ⅰ维甲酸诱导基因Ⅰ(Rig-Ⅰ)
3)retinoic acid-induced geneⅠ维甲酸诱导基因Ⅰ
1.PAMP can be detected by RIG-1(protein encoded byretinoic acid-induced geneⅠ) intracellularly,RIG-1 contains a RNA helicase domain .病毒在复制过程中产生的复制中间体双链RNA作为一个病原相关分子模式,被细胞内具有RNA解旋酶活性的维甲酸诱导基因Ⅰ(RIG-1)编码蛋白检测到。
英文短句/例句
1.Cloning of the Porcine Retinoblastoma-Inhibiting Gene Ⅰ and Its Effect on the Induction of Type Ⅰ Interferon猪维甲酸诱导基因Ⅰ（RIG-Ⅰ）的克隆及其在诱导Ⅰ型干扰素中的作用研究
2.Role of retinoic acid-induced gene Ⅰ signal pathway during West Nile virus infection维甲酸诱导基因Ⅰ信号通路在西尼罗病毒感染中的作用研究
3.Alpha-fetoprotein Gene Expression and Methylation in F9 Cells during Retinoic Acid-induced Differentiation;维甲酸（RA）诱导F9干细胞分化过程中甲胎蛋白基因表达及其甲基化
4.All-trans Retinoic Acid Induced Differentially Genes Expression in Glioma cells全反式维甲酸诱导胶质瘤细胞基因的差异表达
5.Effects of Mutated Presenilin 1 on the Functions of RA-induced PC12 Cells;突变型Presenilin-1基因对全反式维甲酸诱导PC12细胞功能的影响
6.Relationship between gene expression of β-catenin and embryonic neural tube defects induced by Retinoic acid维甲酸诱导胚胎神经管畸形与β-catenin基因表达的关系
7.SEQUENCE ANALYSIS OF GENE MUTATION INDUCED BY GLYCIDYL METHACRYLATE甲基丙烯酸环氧丙酯诱导基因突变的序列分析
8.Study on Resistant Gene of Citrus Induced by Methyl Jasmonate and Scale Insect茉莉酸甲酯和蚧虫诱导柑橘抗性基因的研究
9.The Embryonic Processes and the Expression of the RAR and Hox Gene in Rat Fetuses with Anorectal Malformations Induced by Retinoic Acid;维甲酸诱导大鼠肛门直肠畸形模型演变规律、RAR及Hox基因表达的研究
10.Smad Signaling Mechanism in atRA-induced Cleft Palate;维甲酸诱导腭裂发生的Smad信号机制
11.The Study of Combination of Arsenic Trioxide with All-trans Retinoic Acid on Induction of NF-kb、ROS in NB4 Cells;三氧化二砷联合全反式维甲酸诱导NB4细胞核因子-kb、ROS表达的研究
12.Extrinsic Salicylic and Peroxide Induce Changes in Cytosine Methylation in the Rice Genome;外源水杨酸和过氧化氢诱导水稻基因组发生DNA甲基化变异
13.Phenylhexyl isothiocyanate induces gene p15 demethylation by down-regulating DNA methyltransferases in Molt-4 cells异硫氰酸苯己酯诱导Molt-4细胞p15基因去甲基化及机制研究
14.Study on the Gene Expressed Profile of Di(2-ethylhexyl) Phthalate-induced Brain Tissue in Drosophila Melanogaster;邻苯二甲酸二酯诱导果蝇脑组织基因差异表达谱的研究
15.Retinoic Acid Receptor γ Expression Is Induced in Fibroblast Cell by Vitamin A维生素A诱导成纤维细胞内视黄酸受体γ基因的表达变化研究
16.Expression Analysis of Type Ⅰ hepcidin Gene from Rice Field Eel(Monopterus albus)黄鳝Ⅰ型hepcidin抗菌肽基因的诱导表达分析
17.Calmodulin Kinase Ⅱ Activation of Mitogen-activated Protein Kinase in PC12 Cell Following All-trans Retinoic Acid Treatment;全反式维甲酸诱导PC12细胞凋亡中CaMKⅡ激活MAPK途径
18.Screening of the activity of K562 cell differentiation induced by retinoic acid derivatives维甲酸类衍生物诱导K562细胞分化的活性筛选
相关短句/例句
retinoic acid-induced gene Ⅰ维甲酸诱导基因Ⅰ(Rig-Ⅰ)
3)retinoic acid-induced geneⅠ维甲酸诱导基因Ⅰ
1.PAMP can be detected by RIG-1(protein encoded byretinoic acid-induced geneⅠ) intracellularly,RIG-1 contains a RNA helicase domain .病毒在复制过程中产生的复制中间体双链RNA作为一个病原相关分子模式,被细胞内具有RNA解旋酶活性的维甲酸诱导基因Ⅰ(RIG-1)编码蛋白检测到。
4)retinoic-acid-inducible gene I维甲酸诱导基因I
1.Retinoic-acid-inducible gene I (RIG-I), as a member of theretinoic-acid-inducible gene I(RIG-I)-like receptors (RLRs), plays an important role in the induction of antiviral innate responses including induction of type I IFNs.维甲酸诱导基因I(Retinoic-acid-inducible gene I,RIG-I)作为机体识别病毒的主要免疫识别受体属于维甲酸诱导基因I样受体家族(Retinoic-acid-inducible gene I(RIG-I)-like receptors,RLRs)的主要成员,在机体的抗病毒免疫应答特别是I型干扰素的诱导产生中发挥着重要作用,此外,与细胞的生长调控也有一定的关系,是近年来免疫学基础研究的前沿热点之一。
5)retinoic acid induced gene I (RIG-I)维甲酸诱导基因I(RIG-I)
6)Gene associated with retinoid-interferon-induced mortality-19维甲酸-干扰素诱导的死亡相关基因19
延伸阅读
光诱导型基因传递系统
光诱导型基因传递系统
东京大学（the university of tokyo）研究者的最新报道指出，他们解决了在基因治疗中的最大挑战—将dna安全、有效地转入细胞。他们开发了一种光诱导的基因传递系统（gene delivery system）：将dna分子和光敏感化合物组装进一个简单的结构中，进而使实验操作对细胞的毒害降低到最小。类似dna的大分子进入细胞的主要方法是通过胞吞作用（endocytosis），细胞外膜内陷将化合物单独隔离在一个称作内涵体（endosome）的区室中。光敏分子（photosensitive molecule）在接受光激发的时候帮助dna分子破膜而出。在dna周围包被上这种分子，使得研究者对细胞内其他细胞器和膜结构的破坏降低到最小。
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