糖皮质类固醇激素诱导的肿瘤坏死因子受体家族相关受体,GITR
1)GITR糖皮质类固醇激素诱导的肿瘤坏死因子受体家族相关受体
2)Glucocorticoid-induced tumor necrosis factor receptor糖皮质激素诱导的肿瘤坏死因子受体
1.Objective To explore the change and clinical significance of the expression level of glucocorticoid-induced tumor necrosis factor receptor(GITR) mRNA in peripheral blood mononuclear cells of patients with Graves disease(GD).目的探讨Graves病(GD)患者糖皮质激素诱导的肿瘤坏死因子受体(GITR)mRNA表达水平的变化及意义。
3)human glucocorticoid-inducible tumor necrosis factor receptor人类糖皮质激素诱导的肿瘤坏死因子受体
4)TRAILR肿瘤坏死因子相关凋亡诱导配体受体
1.Objective To investigate the expression of TNF-related apoptosis-inducing ligand(TRAIL) and TNF-related apoptosis-inducing ligand receptor(TRAILR) in thyroid carcinoma.目的研究肿瘤坏死因子相关凋亡诱导配体 (TRAIL)和肿瘤坏死因子相关凋亡诱导配体受体(TRAILR)在甲状腺癌中的表达及意义。
5)Tumor necro sis factor-related apoptosis-induce ligand (TRAIL) recptors肿瘤坏死因子相关诱导凋亡配体受体
英文短句/例句
1.The Expression and Significance of Trail Recepters mRNA in Hepatocellular Carcinoma;肿瘤坏死因子相关诱导凋亡配体受体mRNA在肝癌中的表达及意义
2.The Expression of Hypoxia-inducible Factor 1α and TNF Related Apoptosis Inducing Ligand Receptor in Cervical Cancer and Their Clinical Significance;缺氧诱导因子-1α与肿瘤坏死因子相关诱导凋亡配体受体在宫颈癌组织中的表达及意义
3.Gene Therapy with Tumor Necrosis Factor-related Apoptosis Inducing Ligand Against Glioma;肿瘤坏死因子相关凋亡诱导配体诱导胶质瘤凋亡的研究
4.TRAIL/TRAILR and Pregnancy肿瘤坏死因子相关凋亡诱导配体及其受体与妊娠
5.Tumor Necrosis Factor-related Apoptosis-inducing Ligand Induce Apoptosis in Human Hepatocellular Carcinoma Cell Lines;肿瘤坏死因子相关的凋亡诱导配体诱导肝癌细胞凋亡的实验研究
6.Function of Tumor Related Apoptosis Inducing-ligand to Human Osteosarcoma Cells肿瘤坏死因子相关凋亡诱导配体对人骨肉瘤的作用
7.Treatment of Osteosarcoma with TNF-Related Apoptosis-Inducing Ligand in Nude Mice肿瘤坏死因子相关凋亡诱导配体对裸鼠骨肉瘤的治疗作用
8.Preparation of Monoclonal Antibody against Extracellular Domain of TNF-related Apoptosis-induced Ligand抗肿瘤坏死因子相关凋亡诱导配体细胞外段单克隆抗体的制备
9.PROKARYOTIC EXPRESSION OF MANDARIN FISH TUMOR RELATED APOPTOSIS INDUCING LIGAND GENE鳜肿瘤坏死因子相关的凋亡诱导配体Trail基因的原核表达
10.Experimental Study of TNF-Related Apoptosis-Inducing Ligand on Human Bladder Cancer;肿瘤坏死因子相关诱导凋亡配体对人膀胱癌治疗作用的实验研究
11.An Experimental Study on Treatment of Pancreatic Carcinoma with Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand;肿瘤坏死因子相关凋亡诱导配体治疗胰腺癌的实验研究
12.Study on the Effection of TRAIL on the Leukemic Cell Lines;肿瘤坏死因子相关凋亡诱导配体对白血病细胞影响的研究
13.The expression of TRAIL in psoriatic patients and healthy subjects肿瘤坏死因子相关凋亡诱导配体在寻常型银屑病皮肤中的表达
14.DNA Residue Detection of Recombination Human Tumor Necrosis Factor(TNF)-related Apoptosis-inducing Ligand重组人肿瘤坏死因子相关凋亡诱导配体蛋白的DNA残留量检测
bination therapy of Docetaxel with radiation increases apoptosis of human Hela cell line and expressions of TRAIL and its receptors肿瘤坏死因子相关诱导凋亡配体及其受体在多西紫杉醇对人宫颈癌放疗增敏中的作用
16.Experimental Study of the Apoptosis Effect Induced by TRAIL on Laryngeal Squamous Cell Carcinoma;肿瘤坏死因子相关凋亡诱导配体诱导喉鳞状细胞癌凋亡的实验研究
17.Mechanism Underlying Synergistic Apoptosis-inducing Activity of As_2O_3 Combined with rhTRAIL on Gastric Cancer Cells三氧化二砷与重组人肿瘤坏死因子相关细胞凋亡诱导配体协同诱导胃癌细胞凋亡的机制
18.The Experimental Studies of Tumor Necrosis Factor Related Apotosis-Inducing Ligand(TRAIL) on Proliferation and Apoptosis of Breast Cancer Cell Lines in Vitro;肿瘤坏死因子相关凋亡诱导配体对乳腺癌细胞增殖和凋亡影响的实验研究
相关短句/例句
Glucocorticoid-induced tumor necrosis factor receptor糖皮质激素诱导的肿瘤坏死因子受体
1.Objective To explore the change and clinical significance of the expression level of glucocorticoid-induced tumor necrosis factor receptor(GITR) mRNA in peripheral blood mononuclear cells of patients with Graves disease(GD).目的探讨Graves病(GD)患者糖皮质激素诱导的肿瘤坏死因子受体(GITR)mRNA表达水平的变化及意义。
3)human glucocorticoid-inducible tumor necrosis factor receptor人类糖皮质激素诱导的肿瘤坏死因子受体
4)TRAILR肿瘤坏死因子相关凋亡诱导配体受体
1.Objective To investigate the expression of TNF-related apoptosis-inducing ligand(TRAIL) and TNF-related apoptosis-inducing ligand receptor(TRAILR) in thyroid carcinoma.目的研究肿瘤坏死因子相关凋亡诱导配体 (TRAIL)和肿瘤坏死因子相关凋亡诱导配体受体(TRAILR)在甲状腺癌中的表达及意义。
5)Tumor necro sis factor-related apoptosis-induce ligand (TRAIL) recptors肿瘤坏死因子相关诱导凋亡配体受体
6)DR4肿瘤坏死因子相关凋亡诱导配体受体(DR4)
延伸阅读
糖皮质激素糖皮质激素glucocorticoid简称“皮质激素”。肾上腺束状带细胞所合成和分泌的激素。有氢化可的松和可的松等。主要影响糖和蛋白质的代谢，而对水盐代谢影响较小。且能对抗炎症反应，临床应用较广。其分泌和生成受腺垂体分泌的促肾上腺皮质激素(ACTH）所调节和控制。当机体中糖皮质激素过多时，反过来可以抑制腺体产生ACTH，两者互相影响，维持着动态平衡。一般可的松和氢化可的松可由机体内合成，亦可由人工合成。近年来通过化学方法在可的松和氢化可的松结构上加以改变，合成30多种可的松类衍生物，用于临床者约十余种，如强的松、强的松龙、甲基强的松龙、氟强的松龙、氟羟强的松龙、对氟米松、倍他米松、地塞米松等。临床主要利用它超生理剂量的抗炎、抗过敏、免疫抑制、抗毒和抗休克等药理作用来治疗一系列疾病。主要适合于严重中毒性感染、结缔组织病、过敏性疾病、肾病综合征、血液病、移植排斥反应的预防以及局部外用等。长期大剂量使用可引起类肾上腺皮质功能亢进综合征、骨质稀疏和肌萎缩、类固醇性糖尿病，诱发和加重溃疡和出血、穿孔，并发和加重感染及精神病。停药不合理可致医源性肾上腺皮质功能不全、激素停用后综合征和反跳现象。因此严重精神病、癫痫、活动性消化性溃疡、糖尿病、严重高血压、骨折、创伤修复期、恶病质，抗菌药物不能控制的感染(如水痘、麻疹、霉菌感染等），均应禁用皮质激素。
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